Broselow Cart

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Level 1 pediatric trauma patients are usually an infrequent occurrence, but they are severe and it is important to be prepared for them. Pediatric patients are also more complicated almost all the equipment, medications, and even vital signs parameters are different depending on the age/weight/height of the patient. This makes evaluating and managing a pediatric patient more complex and nuanced. 

 

The Broselow Cart is designed to have the proper equipment by weight range, but can also be used in conjunction with the broselow tape if a weight cannot be obtained.

 Let's set the scene, we get a notification that they're bringing in a 6 month old, unresponsive, 5 minutes out. 

To set up the resus room for this patient, we check that there is an appropriate infant BVM on the Broselow cart, place the backboard on the bed along with the Broselow tape, and crack open the cart.

When you open the drawers of the Broselow cart, it may be overwhelming if you have never seen the contents before. The first drawer is medications that are commonly used in pediatric resuscitations.

 

 

Be aware that both the cardiac arrest epi and the anaphylaxis epi are in here, the beige tan colored box has the cardiac arrest epi which is lower concentration higher volume as the medication is given IV, while the white box with  the pink star has the anaphylaxis epi which is a higher concentration and lower volume as the medication is given IM.

 

Next you open the bottom drawer and take note that the laryngoscope handles and McGill Forceps are in here.

 

Just as you open the pink/red drawer, the patient rolls in with EMS:

6 month old female who just started eating solids, had eggs and peanuts for the 2nd time in her life today. Parent states that the patient had 1 episode of vomiting and thought the patient just ate too much and let the patient rest in the crib. Reports that after completing chores, they went to check on the patient 30 minutes later and noticed her face and body was red, swollen, and she was having difficulty breathing so EMS was called. Upon EMS arrival, patient was given 0.01mg/kg IM epinephrine, about 10 minutes ago. 

 

Upon arrival in the ED, patient appears lethargic, swollen face, wheezing throughout.

Vitals: HR 190bpm, RR 60, BP 60/30, O2 sat 80% on NRB. 

 

While a nurse is attempting to get an IV, you tell another nurse to give the patient another dose of 0.01mg/kg epi, get a second IV and administer IV fluids (20mL/kg or 140mL of normal saline), ask pharmacist to prepare 0.1mcg/kg/min epinephrine drip, and prepare for intubation anticipating a difficult airway, call anesthesia. Ask RT to bag the patient with the infant BVM.

 

You turn around to collect your supplies and the pink/red drawer looks like this:

 

 

Most of the airway equipment is on the left section with the ETTs being stored in the middle. If multiple people are using this cart, it will likely not be organized so just knowing what is in the drawer itself is the most important. 

 

For this patient I would grab an OPA, PEEP valve, the small hyper-angulated rigid stylet and the LoPro S1 blade from the left side of the drawer, and under the BP cuffs to get the 3.5mm uncuffed ETT. As a backup, I will get the 6Fr stylet as well as the miller blade and a blade handle for the most bottom drawer. Also, be sure to check that the suction on the wall is working and get the small Yankauer catheter hooked up. Find the makeshift needle cricothyrotomy kit which includes a 14G angiocath, a 3mL syringe, and adapter to 7.0mm ETT.

 

Next, ask the nurse to draw up ketamine 1.5mg/kg or 10.5mg, use the infant BVM with the PEEP valve to bag the patient, get a repeat set of vitals, and attempt to intubate the patient.

 

We never know what may be rolling through those doors, so it is important to be ready for anything and everything. 

 

For this Trauma Tuesday we will go through a pediatric trauma case based on a case that I was a part of 2+ years ago while also reviewing some trauma reminders and the rest of the Broselow Cart!

6 y/o male brought in to resus 51 as a level 1 trauma, intubated after being rolled over by a car that reversed onto the sidewalk while the patient was crouched down and playing next to the curb. EMS reports that patient was hypoxic and unresponsive, intubated, vitals improved.

Upon arrival in the ED, trauma team and peds ED team at bedside.

Vitals: HR 135bpm, RR bagged at 18, BP 100/60, O2 sat 93% bagged

Airway - intubated, Breathing - bilateral breath sounds, Circulatory - distal pulses intact, tachycardic.

e-FAST negative, Primary survey - large right sided chest wall contusion and ecchymosis, Secondary survey - large ecchymosis on the right upper back.

CXR with large right hemothorax, PXR negative, patient was taken to CT scanner and found with large right hemothorax before going to Peds 31.

Patient's O2 saturation dropping from 93% to 85%, thought it was due to hemothorax, and a right sided chest tube was placed. 

Pause: 

One of the things that we should've done is open the Broselow cart prior to when the patient initially arrived in Resus 51, I think we estimated his weight to be 20kg by the Broselow tape but did not open the cart since the patient was already intubated. Even though it was not used in this case, we should get in the practice of opening the cart whenever a pediatric resus patient arrives.

One of the barriers to opening the cart is not knowing what is in the cart, so the POTD from yesterday gave an overview, and here are the rest of the drawers. Everything contained in each drawer is the same, just different sizes for the different estimated weights of the patient according to the Broselow tape.

In addition, all pediatric trauma patients need a trauma observation order, a recorded weight, and height, so there is a new tall block in peds resus 30 with a tape measure attached to measure our patient's height:

Here is the rest of the Broselow Cart:

 

 

This patient got a 24Fr Chest tube in the right chest with about 200cc of output, but the O2 saturation was not improving and patient desaturated to the low 80s. A CXR was obtained to confirm chest tube placement but it also revealed that the ETT tube was too high.

Upon review of the first CXR, it seems that the ETT tube still seemed slightly high but probably was in place and got dislodged as the patient was moved multiple times now. The peds attending and senior EM resident utilized video laryngoscopy and found the balloon above the cords. The patient started to desaturate to the 70s, the dislodged ETT was removed and they bagged the patient. Patient's O2 saturation improved to high 80s/low 90s and they reintubated the patient. After intubation, the patient's right chest tube put out another 200cc of blood and continuing, so the decision was made to take the patient to the OR and given MTP. In the OR, the patient arrested while they were performing a thoracotomy but they were unable to achieve ROSC.

Patient had a bronchial injury causing the large hemothorax and even with MTP and making it to the OR the patient died. It was a reminder that pediatric patients are able to compensate for the most part until they are not. It is a constant reminder that we have to stay vigilant and be prepared. We check the resus bays and review airway plans every shift in order to get ahead of it. Now let's keep the Broselow cart in mind for pediatric resuscitations!

Takeaways:

 

  • Every pediatric trauma patient needs a recorded height, weight, and a trauma observation order.

  • Open the Broselow cart before the patient arrives to review your equipment.

  • Crack open the Broselow cart for any pediatric resuscitations!

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Analgesia for Acute Anterior Shoulder Dislocation Reduction

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Hello all,

For this Trauma Tuesday imagine you're a 1945 WWII nurse transported back in time to 1743 Scotland and a Jacobite warrior dislocates his shoulder. You need to reduce it but all they have for pain control is Scottish whiskey and if you're lucky, something called Laudanum. (So goes the plot of Outlander)

Laudanum is a mixture of opium and ethanol which is old school procedural sedation and analgesia (PSA) that is equivalent to fentanyl and versed that we use in the modern day. It is definitely a tried-and-true combo that has crossed into the 21st century but how does it fair against intra-articular lidocaine (IAL)?

First off, anterior shoulder dislocations is the most common shoulder dislocation ~95%, posterior shoulder dislocation ~5%, and inferior shoulder dislocation <1%.

In these images, the red arrow is pointing at the glenoid fossa and the blue arrow is pointing at the humeral head which should be in contact with the glenoid fossa.

To inject Intra-articular lidocaine first locate the landmarks, since this is an anterior shoulder dislocation the posterior approach to the glenohumeral joint and subacromial space may be the easiest as that space is widened due to the dislocation. 

Have the patient sit with their arm resting at their side if possible. Palpate the posterior indent between the head of the humerus and acromion, using a 18G needle with 20 mL of 1% lidocaine, insert the needle 2-3cm inferior and medial to the posterolateral corner of the acromion and direct it anteriorly towards the coracoid process when aiming for the glenohumeral joint. When aiming for the subacromial space, direct it laterally while aiming for the anterolateral acromion corner. However, any space between these two areas will be sufficient for an intra-articular block with a dislocation. An 18G needle should sink into the space and the plunger should push with no resistance if you are in the correct space. 

One study did a systematic review and meta-analysis of 12 RCTs comparing IAL to PSA (with any agent) for closed reduction of acute anterior shoulder dislocation for patients > 15 years of age.

Results:

  • No difference in reduction success between IAL and PSA. (83.8% vs. 91.4%)

  • Fewer adverse events occurred in the IAL group compared to the PSA group. (1.3% vs. 20.8%) 

  • Mean ED length of stay was significantly shorter in the IAL group compared to the PSA group. (mean difference = − 1.48 h)

  • No difference in pain score after anesthesia and before reduction in the IAL group compared to the PSA group. (mean difference = − 0.04)

  • Procedural time was significantly longer in the IAL group compared to PSA (mean difference = 8 min)

  • No statistically significant difference in ease of reduction in the IAL group compared to PSA. (54.5% vs. 71.8%)

  • Patient satisfaction was significantly decreased in the IAL group compared to PSA. (70.5% vs. 90.4%)

TL;DR

IAL seems to have similar effectiveness as PSA in the successful reduction of anterior shoulder dislocations in the ED with fewer adverse events, shorter ED length of stay, and no difference in pain scores, or ease of reduction. However, IAL had longer procedural times and decreased patient satisfaction which is something to consider. Nonetheless, IAL may be an effective alternative to procedural sedation for reducing anterior shoulder dislocations, particularly when IV sedation is contraindicated or not feasible.

https://rebelem.com/intra-articular-lidocaine-vs-procedural-sedation-and-analgesia-for-closed-reduction-of-acute-anterior-shoulder-dislocation/

https://wikem.org/wiki/Shoulder_dislocation

https://www.shoulderdoc.co.uk/article/1485

Sithamparapillai, A, Grewal, K,Thompson, C, Walsh, C, McLeod, S. Intra-Articular lidocaine versus intravenous sedation for closed reduction of acute anterior shoulder dislocation in the Emergency Department: a systematic review and meta-analysis. Canadian Journal of Emergency medicine. October 2022. PMID: 36181665

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Pong Pong Tree

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Hello everyone!

For those of you who don't know me, I am Karen, a the third year resident who will be staying on as one of the ultrasound fellows next year (along with the amazing Dan Ye, who can forget his great recaps of The Pitt), I'm also one of the four chiefs following our previous admin resident and chief Kaitlyn DeStefano (whose POTDs made us laugh and cry). I have big shoes to fill with these two tough acts to follow, but I will work hard to bring fun and educational POTDs!

Continuing on The Pitt bandwagon, I love watching shows, from romances and drama, to murder mysteries and thrillers, to Kdramas and anime; I am a huge consumer of media for better or for worse. Some of my favorites are Outlander, Downton Abbey, Dark, Black Mirror, Squid Game, Attack on Titan, When Life Gives You Tangerines, of course The Pitt and so many more...

Anyways, since yesterday was the finale of HBO's "White Lotus" I wanted to talk about the Pong pong tree. (Spoilers!)

The Pong Pong tree (Cerbera odollam)—also known as the “suicide tree” is native to India and Southeast Asia. This plant contains cerberin, a cardiac glycoside similar to digoxin that disrupts cardiac activity by inhibiting the Na⁺/K⁺- ATPase pump, which can lead to fatal arrhythmias. Cerberin poisoning is difficult to detect and has been used in homicides due to its tasteless nature and delayed onset. There is no specific antidote, though digoxin-specific Fab fragments (DIGIFab) has been used.

There has been only 2 known cases of C. odollam poisoning and 1 fatality reported in the USA:

A 22-year-old pre-operative transgender man-to-woman patient ingested seeds from the Cerbera odollam tree as a suicide attempt. She presented to the emergency department with nausea, vomiting, chest pain, and dizziness approximately 7 hours after ingesting the seeds that she bought online after reading about suicide. 

Initial electrocardiogram (ECG) showed second-degree heart block with 2:1 atrioventricular (AV) conduction and ST-segment depression with biphasic T-waves and initial serum potassium was 5.2mEq/L:

So the team administered atropine, digoxin-Fab fragments, and supportive care with improvement to sinus rhythm with first-degree AV block with persistent ST-segment depression and biphasic T-waves:

However, 2 hours later her condition deteriorated, progressing to high-degree AV block:

And subsequent cardiac arrest 30 minutes later with repeat K of 5.7mEq/L.

Despite additional doses of digoxin-specific antibody fragments (20 vials in total), lipid emulsion 20% (100 ml), right femoral CVC, and 2 hours of aggressive ACLS resuscitative efforts the patient was pronounced dead approximately 12 hours post-ingestion.

This case delineates the severe toxicity associated with C. odollam seed ingestion and highlights the challenges in managing such poisonings, even with advanced supportive measures. We should all be aware of the potential morbidity and mortality linked to this plant toxin and be prepared for aggressive resuscitative interventions.

Although the USA has only had 2 known cases of C. odollam poisoning, will a television show introducing this plant open the gates for more? Hopefully not, but as always, we will be prepared.

Misek R, Allen G, LeComte V, Mazur N. Fatality Following Intentional Ingestion of Cerbera odollam Seeds. Clin Pract Cases Emerg Med. 2018 Jun 12;2(3):223-226. doi: 10.5811/cpcem.2018.5.38345. PMID: 30083638; PMCID: PMC6075506. https://pmc.ncbi.nlm.nih.gov/articles/PMC6075506/

Karen Wong, MD

Emergency Medicine Chief Resident

KaWong@maimo.org

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