POTD: Traumatic brain injuries (part 2)

Welcome to Trauma Tuesday! This will be a continuation of my first POTD on TBIs, but today, we will be talking more about the public health implications of TBIs.

According to the CDC, there were over 210,000 TBI-related hospitalizations and a little under 70,000 TBI-related deaths in 2021 in the United States. In addition to direct health-related statistics, there are many long-term effects of TBIs that have an impact on the individual's quality of life and on society as a whole. For example, patients with a history of TBIs are more likely to have difficulties with finding work and holding down a job, dysregulated behavior that can lead to challenges in social relationships, and substance use disorders.

Additionally, vast health disparities exist on who suffers from a TBI. Statistics show that adults aged 75 years or older, racial/ethnic minorities, veterans, people who are incarcerated, people experiencing homelessness, and victims of domestic violence are both more likely to get a TBI and to suffer worse long-term consequences from it. Many of the people who fall under the above mentioned categories also have more difficulty accessing healthcare, which can put even more barriers in their road to recovery, as some patients require long-term services such as physical/occupational therapy and mental health support. 

Given everything I mentioned above, it is tremendously important that we do our best in the acute management of head injuries in the ED. However, perhaps even more important is TBI prevention before the primary injury ever happens. While our time and our resources are limited in the ED, we have the opportunity to do some quick education with our patients and their families on how to prevent (more) TBIs - everything from wearing helmets to assessing the fall risk of a patient who may have mobility issues. While we can't control what happens to our patients outside of the ED, we can at least take a few minutes to talk to them about this topic in hopes of saving them a (potential) lifetime of further complications.

Resources:

https://emcrit.org/ibcc/tbi/#coagulation_management

https://www.emdocs.net/neurotrauma-resuscitation-pearls-pitfalls/

https://www.saem.org/about-saem/academies-interest-groups-affiliates2/cdem/for-students/online-education/m4-curriculum/group-m4-trauma/closed-head-injury

https://www.cdc.gov/traumatic-brain-injury/health-equity/

Thurman DJ, Alverson C, Dunn KA, Guerrero J, Sniezek JE. Traumatic brain injury in the United States: A public health perspective. J Head Trauma Rehabil. 1999;14(6):602-615. doi:10.1097/00001199-199912000-00009

Peterson AB, Zhou H, Thomas KE. Disparities in traumatic brain injury-related deaths-United States, 2020. J Safety Res. 2022 Dec;83:419-426. doi: 10.1016/j.jsr.2022.10.001. Epub 2022 Oct 18. PMID: 36481035; PMCID: PMC9795830.

Wilson MH. Traumatic brain injury: an underappreciated public health issue. Lancet Public Health. 2016;1(2):e44. doi:10.1016/S2468-2667(16)30022-6


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POTD: Traumatic brain injuries (part 1)

Hi everyone!

My name is Nicky, and I will be your new admin resident for this block. You all know what that means - it's time for me to be on my soapbox for the next 4 weeks.

I'm going to start off my month by discussing traumatic brain injuries (TBIs), and as public health is a big interest of mine, I'm also going to discuss their impact on public health (later, in part 2).

What is a TBI, and what leads to worse outcomes?


A TBI is any kind of trauma to the brain - some may be mild, like a bump to the head, and some may be severe, such as a gunshot wound or a high mechanism fall. Morbidity and mortality from TBIs can come from primary injury, which is neuronal damage directly due to the traumatic event at the time of the traumatic event, or secondary injury, which is damage due to sequalae of the traumatic event.


Some things that may lead to secondary injury include:

- Edema and elevated ICP

- Hypotension

- Hypoxia

- Hyperoxia

- Fever

- Seizures

Given that the primary injury has already occurred by the time the patient is in the ED, our goal is to prevent secondary injury. 

What can we do to optimize patient outcomes?

Studies have shown that goals for physiologic parameters are, more or less, the ranges of normal that we think of in the ED:

- SpO2 > 94% but less than 100%

- SBP > 100

- pH 7.35-7.45

- Glucose 80-180

And also...

- ICP (intracranial pressure) < 22mmHg

- CPP (cerebral perfusion pressure) > 60 mmHg

To measure ICP accurately, it requires a monitor placed directly in the ventricle, so oftentimes we do not have this in the ED. However, there are several signs we can look for of increased ICP, including the Cushing reflex (hypertension, bradycardia, and respiratory irregularity). Other signs include a fixed and dilated pupil in uncal herniation and bilateral pinpoint pupils in central transtentorial herniation and in cerebellotonsillar herniation. 

Additionally, on imaging, if you see a significant ICH especially with midline shift, it's important to do frequent assessments of the patient as they are at high risk of increased ICP. 

I won't discuss the other parameters as the management is typically self-explanatory, but specifically for elevated ICP, there are several things that can be done in the ED:

- Elevating the head of the bed to 30 degrees

- Mannitol or hypertonic saline 

- Hyperventilation

- Antiemetics as vomiting will increase ICP

and ultimately, neurosurgical consultation as these patients may require surgical decompression.

And that's a quick and not at all comprehensive overview of TBIs and ED management. To keep things concise, I'll talk about public health implications in my next POTD. Stay tuned!

Resources:

https://emcrit.org/ibcc/tbi/#coagulation_management

https://www.emdocs.net/neurotrauma-resuscitation-pearls-pitfalls/

https://www.saem.org/about-saem/academies-interest-groups-affiliates2/cdem/for-students/online-education/m4-curriculum/group-m4-trauma/closed-head-injury

https://www.cdc.gov/traumatic-brain-injury/health-equity/

Thurman DJ, Alverson C, Dunn KA, Guerrero J, Sniezek JE. Traumatic brain injury in the United States: A public health perspective. J Head Trauma Rehabil. 1999;14(6):602-615. doi:10.1097/00001199-199912000-00009

Peterson AB, Zhou H, Thomas KE. Disparities in traumatic brain injury-related deaths-United States, 2020. J Safety Res. 2022 Dec;83:419-426. doi: 10.1016/j.jsr.2022.10.001. Epub 2022 Oct 18. PMID: 36481035; PMCID: PMC9795830.

Wilson MH. Traumatic brain injury: an underappreciated public health issue. Lancet Public Health. 2016;1(2):e44. doi:10.1016/S2468-2667(16)30022-6

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"Abscessed" with Bowel POCUS: Diverticulitis

HPI: 42 yo male with no PMH presenting for abdominal pain x 2 days. His physical exam was significant for LLQ tenderness with guarding and rebound.

POCUS showed (see video):

We initially thought the outpouching connected to the abscess was a diverticulum but on further review, it’s more likely to be a loop of bowel given its size.

What a diverticulum should look like:


CT scan for reference:


Diagnosing Diverticulitis on POCUS

  • Use curvilinear vs linear probe

  • Start at maximal point of pain > lawnmower technique

  • #1: Find diverticula

    • Looks like outpouching attached to loop of bowel

  • Secondary findings:

    • Bowel wall diameter >5 mm

    • Prominent, fluid-filled bowel loops

    • Pericolic fluid collections

    • Increased pericolic fat (hyperechoic fat anterior to diverticula)

    • Intraabdominal abscesses

Case conclusion: CTAP showed perforated diverticulitis with multiple intraabdominal abscess. Patient was taken for IR drainage with feculent/purulent drainage noted. Patient is still doing well on surgical service.

References

  1. https://coreultrasound.com/diverticulitis/

  2. https://www.ultrasoundgel.org/posts/SFPsfN9yJ-9uSp640QlWtg

  3. https://www.ultrasoundcases.info/diverticulosis---diverticulitis-531/